ISSN: 2578-6571
Home / Browse Journals & Books / Journal of Brain Disorders / Archive / Volume 4, Issue 1
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Research Article Pages 92-100
Abstract: In order to support patients suffering from Parkinson’s disease caused by irregular aggregation of alpha-synuclein in the brain, an economically affordable and diagnostically effective modality such as magnetic induction tomographyis proposed to differentiate normal and Parkinson’s disease brain tissues based on imaging. MIT technology has been applied in various medical fields due to its advancement in the diagnosis of diseases. It is sensitive to conductivity, permittivity, and permeability of biological tissue.
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Review Article Pages 82-91
Abstract: Endothelial cells exhibit a variety of structural and functional changes when they come into contact with normal laminar flow. In response to laminar shear stress, endothelial cells modify their potassium ion channels, go through cytoskeletal rearrangements and shape modifications and create prostacyclin. In cerebral arteries, aneurysmal dilatation most frequently starts at locations with substantial wall shear stress, Which include arterial bifurcations and vascular branch sites, where blood flow abruptly switches to turbulent flow.
Research Article Pages 73-81
Abstract: Vascular smooth muscles play a vital role in the growth and propagation of cerebral aneurysms. These highly dynamic cells, characterized by a high degree of plasticity, show structural and functional modifications in response to external environmental influences. Following sustained endothelial injury, vascular smooth muscles avail a new functional capability characterized by proinflammatory, and promatrix remodeling abilities, losing the normal contractile and synthetic ability.
Research Article Pages 65-72
Abstract: Bacterial Meningitis is an acute purulent infection of the leptomeninges in the subarachnoid space, most commonly caused by Streptococcus pneumonia, Neisseria meningitidis, and Haemophilus influenzae. A variety of complications orig- inate from a primary focus of bacterial meningitis, but none as severe as cerebrovascular complications. The basis for the development of post-infective cerebrovascular sequelae following an attack of bacterial meningitis remains largely speculative; however several studies suggest a derangement of the coagulative system in evoking such an outcome. A primary derangement of the coagulative status stems from an unbalance between the activation of coagulative process- es and an inhibition of anticoagulant processes, resulting in a net heightened activation of coagulation.
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